{"id":7969,"date":"2026-04-21T13:29:28","date_gmt":"2026-04-21T13:29:28","guid":{"rendered":"https:\/\/bristishpharmacy.co.uk\/?post_type=product&#038;p=7969"},"modified":"2026-09-16T11:09:25","modified_gmt":"2026-09-16T11:09:25","slug":"oximorfona","status":"publish","type":"product","link":"https:\/\/bristishpharmacy.co.uk\/es\/shop\/oximorfona\/","title":{"rendered":"Oxymorphon"},"content":{"rendered":"<h1>\u00a0<\/h1>\n<div class=\"container\">\n<div id=\"model-response-message-contentr_ac824f912d14e499\" class=\"markdown markdown-main-panel md-content enable-luminous-fast-follows enable-updated-hr-color stronger tutor-markdown-rendering\" dir=\"ltr\" aria-live=\"polite\">\n<h3 data-path-to-node=\"3\">Clinical Monograph: Oxymorphone<\/h3>\n<h2 data-path-to-node=\"4\">1. Classification and Chemical Overview<\/h2>\n<p data-path-to-node=\"5\">Oxymorphone is a phenanthrene-derivative opioid alkaloid. Chemically designated as 4,5$\\alpha$-epoxy-3,14-dihydroxy-17-methylmorphinan-6-one hydrochloride, it is a primary active metabolite of oxycodone as well as a standalone pharmaceutical entity. Under the Anatomical Therapeutic Chemical (ATC) system, oxymorphone is indexed under N02AA05.<\/p>\n<p data-path-to-node=\"6\">Oxymorphone is classified internationally as a highly strictly Controlled Substance (e.g., Schedule II under the US Controlled Substances Act; Schedule 2 \/ Class A Controlled Drug in the UK) due to its high potency, profound abuse liability, and physical dependence risk. It is regulated strictly as a Prescription Only Medicine (POM).<\/p>\n<h2 data-path-to-node=\"7\">2. Mechanism of Action and Pharmacodynamics<\/h2>\n<p data-path-to-node=\"8\">Oxymorphone functions as a full, high-affinity agonist at central nervous system opioid receptors:<\/p>\n<ul data-path-to-node=\"9\">\n<li>\n<p data-path-to-node=\"9,0,0\"><b data-path-to-node=\"9,0,0\" data-index-in-node=\"0\"><span class=\"math-inline\" data-math=\"\\mu\" data-index-in-node=\"0\"><span class=\"katex\"><span class=\"katex-html\" aria-hidden=\"true\"><span class=\"base\"><span class=\"mord mathnormal\">\u03bc<\/span><\/span><\/span><\/span><\/span>-Opioid Receptor Agonism:<\/b> Binds with high selectivity and affinity to central <span class=\"math-inline\" data-math=\"\\mu\" data-index-in-node=\"81\"><span class=\"katex\"><span class=\"katex-html\" aria-hidden=\"true\"><span class=\"base\"><span class=\"mord mathnormal\">\u03bc<\/span><\/span><\/span><\/span><\/span>-opioid receptors (<span class=\"math-inline\" data-math=\"\\mu\" data-index-in-node=\"103\"><span class=\"katex\"><span class=\"katex-html\" aria-hidden=\"true\"><span class=\"base\"><span class=\"mord mathnormal\">\u03bc<\/span><\/span><\/span><\/span><\/span>-OR), inhibiting adenylyl cyclase, hyperpolarizing neurons, and suppressing the transmission of ascending nociceptive signals in the spinal cord dorsal horn and brainstem.<\/p>\n<\/li>\n<li>\n<p data-path-to-node=\"9,1,0\"><b data-path-to-node=\"9,1,0\" data-index-in-node=\"0\">Analgesic Potency:<\/b> Possesses a significantly higher intrinsic analgesic potency compared to morphine (roughly 3 times more potent orally and up to 10 times more potent parenterally).<\/p>\n<\/li>\n<\/ul>\n<h2 data-path-to-node=\"10\">3. Approved Clinical Indications and Dosing Scope<\/h2>\n<p data-path-to-node=\"11\">Licensing for oxymorphone includes:<\/p>\n<ul data-path-to-node=\"12\">\n<li>\n<p data-path-to-node=\"12,0,0\"><b data-path-to-node=\"12,0,0\" data-index-in-node=\"0\">Moderate to Severe Acute and Chronic Pain:<\/b> Management of moderate to severe pain where alternative treatment options (such as non-opioid analgesics or lower-potency opioids) are inadequate, and where around-the-clock continuous opioid therapy is required.<\/p>\n<\/li>\n<\/ul>\n<p data-path-to-node=\"13\"><i data-path-to-node=\"13\" data-index-in-node=\"0\">Dosing &amp; Administration Regimen:<\/i><\/p>\n<ul data-path-to-node=\"14\">\n<li>\n<p data-path-to-node=\"14,0,0\"><b data-path-to-node=\"14,0,0\" data-index-in-node=\"0\">Formulation Variances:<\/b> Available as immediate-release (IR) oral tablets and extended-release (ER) tablets designed for prolonged 12-hour coverage.<\/p>\n<\/li>\n<li>\n<p data-path-to-node=\"14,1,0\"><b data-path-to-node=\"14,1,0\" data-index-in-node=\"0\">Titration Protocol:<\/b> Dosage must be individualized based on pain severity, prior opioid exposure, and patient risk factors. Initiation in opioid-naive patients carries severe risks of fatal overdose.<\/p>\n<\/li>\n<li>\n<p data-path-to-node=\"14,2,0\"><b data-path-to-node=\"14,2,0\" data-index-in-node=\"0\">Administration Integrity (Extended-Release):<\/b> ER tablets must be swallowed whole with water on an empty stomach (food significantly increases peak plasma concentrations). <b data-path-to-node=\"14,2,0\" data-index-in-node=\"170\">Do not cut, crush, chew, or dissolve<\/b> ER tablets, as destruction of the polymer delivery matrix causes immediate drug dumping and lethal overdose.<\/p>\n<\/li>\n<\/ul>\n<h2 data-path-to-node=\"15\">4. Pharmacokinetic Profile and Metabolic Fate<\/h2>\n<ul data-path-to-node=\"16\">\n<li>\n<p data-path-to-node=\"16,0,0\"><b data-path-to-node=\"16,0,0\" data-index-in-node=\"0\">Absorption:<\/b> Oral bioavailability of immediate-release oxymorphone is low (~10%) due to extensive first-pass hepatic metabolism. Food intake increases systemic absorption significantly (up to 50% for IR and over 100% for certain ER formulations). Peak plasma concentrations occur within 0.5 to 1.5 hours for IR and 2 to 3 hours for ER.<\/p>\n<\/li>\n<li>\n<p data-path-to-node=\"16,1,0\"><b data-path-to-node=\"16,1,0\" data-index-in-node=\"0\">Distribution:<\/b> Rapidly distributes across tissue compartments. Plasma protein binding is low to moderate (~10% to 12%).<\/p>\n<\/li>\n<li>\n<p data-path-to-node=\"16,2,0\"><b data-path-to-node=\"16,2,0\" data-index-in-node=\"0\">Biotransformation:<\/b> Extensively metabolized in the liver primarily via non-CYP pathways:<\/p>\n<ul data-path-to-node=\"16,2,1\">\n<li>\n<p data-path-to-node=\"16,2,1,0,0\"><b data-path-to-node=\"16,2,1,0,0\" data-index-in-node=\"0\">Glucuronidation (Major):<\/b> Metabolized via UGT2B7 to <b data-path-to-node=\"16,2,1,0,0\" data-index-in-node=\"51\">oxymorphone-3-glucuronide<\/b> (inactive metabolite).<\/p>\n<\/li>\n<li>\n<p data-path-to-node=\"16,2,1,1,0\"><b data-path-to-node=\"16,2,1,1,0\" data-index-in-node=\"0\">Reduction (Minor):<\/b> Converted to 6-<span class=\"math-inline\" data-math=\"\\alpha\" data-index-in-node=\"34\"><span class=\"katex\"><span class=\"katex-html\" aria-hidden=\"true\"><span class=\"base\"><span class=\"mord mathnormal\">\u03b1<\/span><\/span><\/span><\/span><\/span>-oxymorphol. Oxymorphone does not undergo significant Cytochrome P450-mediated metabolism, reducing certain drug-drug interaction liabilities.<\/p>\n<\/li>\n<\/ul>\n<\/li>\n<li>\n<p data-path-to-node=\"16,3,0\"><b data-path-to-node=\"16,3,0\" data-index-in-node=\"0\">Elimination:<\/b> Excreted predominantly via the kidneys in urine as conjugated metabolites and unchanged drug (&lt; 1%). The terminal elimination half-life (<span class=\"math-inline\" data-math=\"t_{1\/2}\" data-index-in-node=\"150\"><span class=\"katex\"><span class=\"katex-html\" aria-hidden=\"true\"><span class=\"base\"><span class=\"mord\"><span class=\"mord mathnormal\">t<\/span><span class=\"msupsub\"><span class=\"vlist-t vlist-t2\"><span class=\"vlist-r\"><span class=\"vlist\"><span class=\"\"><span class=\"sizing reset-size6 size3 mtight\"><span class=\"mord mtight\">1\/2<\/span><\/span><\/span><\/span><span class=\"vlist-s\">\u200b<\/span><\/span><\/span><\/span><\/span><\/span><\/span><\/span><\/span>) averages 7 to 10 hours for extended-release formulations.<\/p>\n<\/li>\n<\/ul>\n<h2 data-path-to-node=\"17\">5. Physiological Effects and Adverse Event Spectrum<\/h2>\n<p data-path-to-node=\"18\">Oxymorphone depresses central nervous system functions, blunts respiratory drive, slows gastrointestinal motility, and alters vascular tone.<\/p>\n<h3 data-path-to-node=\"19\">Adverse Drug Reaction Spectrum<\/h3>\n<ul data-path-to-node=\"20\">\n<li>\n<p data-path-to-node=\"20,0,0\"><b data-path-to-node=\"20,0,0\" data-index-in-node=\"0\">Very Common (<span class=\"math-inline\" data-math=\"\\ge 1\/10\" data-index-in-node=\"13\"><span class=\"katex\"><span class=\"katex-html\" aria-hidden=\"true\"><span class=\"base\"><span class=\"mrel\">\u2265<\/span><\/span><span class=\"base\"><span class=\"mord\">1\/10<\/span><\/span><\/span><\/span><\/span>):<\/b> Constipation, nausea, somnolence, dizziness, vomiting, pruritus, headache.<\/p>\n<\/li>\n<li>\n<p data-path-to-node=\"20,1,0\"><b data-path-to-node=\"20,1,0\" data-index-in-node=\"0\">Common (<span class=\"math-inline\" data-math=\"\\ge 1\/100\" data-index-in-node=\"8\"><span class=\"katex\"><span class=\"katex-html\" aria-hidden=\"true\"><span class=\"base\"><span class=\"mrel\">\u2265<\/span><\/span><span class=\"base\"><span class=\"mord\">1\/100<\/span><\/span><\/span><\/span><\/span> to <span class=\"math-inline\" data-math=\"&lt;1\/10\" data-index-in-node=\"21\"><span class=\"katex\"><span class=\"katex-html\" aria-hidden=\"true\"><span class=\"base\"><span class=\"mrel\">&lt;<\/span><\/span><span class=\"base\"><span class=\"mord\">1\/10<\/span><\/span><\/span><\/span><\/span>):<\/b> Dry mouth, insomnia, anxiety, confusion, fatigue, excessive sweating, hyperhidrosis, dyspepsia, abdominal pain, diarrhea, urinary retention.<\/p>\n<\/li>\n<li>\n<p data-path-to-node=\"20,2,0\"><b data-path-to-node=\"20,2,0\" data-index-in-node=\"0\">Uncommon (<span class=\"math-inline\" data-math=\"\\ge 1\/1,000\" data-index-in-node=\"10\"><span class=\"katex\"><span class=\"katex-html\" aria-hidden=\"true\"><span class=\"base\"><span class=\"mrel\">\u2265<\/span><\/span><span class=\"base\"><span class=\"mord\">1\/1<\/span><span class=\"mpunct\">,<\/span><span class=\"mord\">000<\/span><\/span><\/span><\/span><\/span> to <span class=\"math-inline\" data-math=\"&lt;1\/100\" data-index-in-node=\"25\"><span class=\"katex\"><span class=\"katex-html\" aria-hidden=\"true\"><span class=\"base\"><span class=\"mrel\">&lt;<\/span><\/span><span class=\"base\"><span class=\"mord\">1\/100<\/span><\/span><\/span><\/span><\/span>):<\/b> Orthostatic hypotension, palpitations, flushing, syncope, paresthesia, visual disturbances, urticaria, rash.<\/p>\n<\/li>\n<li>\n<p data-path-to-node=\"20,3,0\"><b data-path-to-node=\"20,3,0\" data-index-in-node=\"0\">Rare \/ Severe (&lt;1\/1,000):<\/b> Life-threatening respiratory depression, apnea, circulatory depression, anaphylactoid reactions, bronchospasm, paralytic ileus, physical dependence, and severe withdrawal syndrome.<\/p>\n<\/li>\n<\/ul>\n<h2 data-path-to-node=\"21\">6. Contraindications, Drug Interactions, and Clinical Precautions<\/h2>\n<h3 data-path-to-node=\"22\">Contraindicaciones<\/h3>\n<ul data-path-to-node=\"23\">\n<li>\n<p data-path-to-node=\"23,0,0\">Known hypersensitivity to oxymorphone or formulation excipients.<\/p>\n<\/li>\n<li>\n<p data-path-to-node=\"23,1,0\">Significant respiratory depression or acute severe bronchial asthma in an unmonitored setting.<\/p>\n<\/li>\n<li>\n<p data-path-to-node=\"23,2,0\">Known or suspected gastrointestinal obstruction, including paralytic ileus.<\/p>\n<\/li>\n<li>\n<p data-path-to-node=\"23,3,0\">Moderate-to-severe hepatic impairment (due to markedly increased systemic exposure and reduced clearance).<\/p>\n<\/li>\n<li>\n<p data-path-to-node=\"23,4,0\">Concomitant use with Monoamine Oxidase Inhibitors (MAOIs) or within 14 days of their discontinuation.<\/p>\n<\/li>\n<\/ul>\n<h3 data-path-to-node=\"24\">Key Drug Interactions<\/h3>\n<ul data-path-to-node=\"25\">\n<li>\n<p data-path-to-node=\"25,0,0\"><b data-path-to-node=\"25,0,0\" data-index-in-node=\"0\">CNS Depressants, Benzodiazepines, Alcohol, &amp; Other Opioids:<\/b> Co-administration produces profound, synergistic central nervous system and respiratory depression, dramatically increasing the risk of fatal overdose, coma, and respiratory arrest.<\/p>\n<\/li>\n<li>\n<p data-path-to-node=\"25,1,0\"><b data-path-to-node=\"25,1,0\" data-index-in-node=\"0\">Alcohol Interaction with Extended-Release:<\/b> Co-ingestion of alcohol with extended-release oxymorphone formulations can cause rapid dissolution and dose dumping of the active drug, leading to fatal plasma concentrations.<\/p>\n<\/li>\n<\/ul>\n<h3 data-path-to-node=\"26\">Clinical Precautions and Monitoring<\/h3>\n<ul data-path-to-node=\"27\">\n<li>\n<p data-path-to-node=\"27,0,0\"><b data-path-to-node=\"27,0,0\" data-index-in-node=\"0\">Boxed Warning (Addiction, Abuse, and Misuse):<\/b> Oxymorphone exposes patients to risks of addiction, abuse, and misuse, which can lead to overdose and death. Assess each patient&#8217;s risk prior to prescribing and monitor regularly.<\/p>\n<\/li>\n<li>\n<p data-path-to-node=\"27,1,0\"><b data-path-to-node=\"27,1,0\" data-index-in-node=\"0\">Boxed Warning (Life-Threatening Respiratory Depression):<\/b> Serious, life-threatening, or fatal respiratory depression may occur. Monitor patients closely, especially during initiation or following dose titration.<\/p>\n<\/li>\n<li>\n<p data-path-to-node=\"27,2,0\"><b data-path-to-node=\"27,2,0\" data-index-in-node=\"0\">Dependence &amp; Withdrawal:<\/b> Prolonged continuous administration establishes physical and psychological dependence. Abrupt cessation triggers a severe withdrawal syndrome. Gradual medical tapering is mandatory.<\/p>\n<\/li>\n<\/ul>\n<\/div>\n<\/div>\n\n    <div class=\"xs_social_share_widget xs_share_url after_content \t\tmain_content  wslu-style-1 wslu-share-box-shaped wslu-fill-colored wslu-none wslu-share-horizontal wslu-theme-font-no wslu-main_content\">\n\n\t\t\n        <ul>\n\t\t\t        <\/ul>\n    <\/div>","protected":false},"excerpt":{"rendered":"<p data-path-to-node=\"1\">Oxymorphone is a potent, semi-synthetic pure opioid agonist derived from morphine. It is indicated for the management of moderate to severe acute and chronic pain requiring continuous, round-the-clock opioid analgesia.<\/p>\n<h3 data-path-to-node=\"3\">\u00a0<\/h3>","protected":false},"featured_media":7973,"comment_status":"closed","ping_status":"closed","template":"","meta":{"postBodyCss":"","postBodyMargin":[],"postBodyPadding":[],"postBodyBackground":{"backgroundType":"classic","gradient":""}},"product_brand":[],"product_cat":[15],"product_tag":[230],"class_list":["post-7969","product","type-product","status-publish","has-post-thumbnail","product_cat-uncategorized","product_tag-oxymorphon","instock","shipping-taxable","purchasable","product-type-simple"],"yoast_head":"<!-- This site is optimized with the Yoast SEO Premium plugin v26.5 (Yoast SEO v28.5) - https:\/\/yoast.com\/product\/yoast-seo-premium-wordpress\/ -->\n<title>Oxymorphon - British Pharmacy<\/title>\n<meta name=\"description\" content=\"Oxymorphone is a potent semi-synthetic opioid analgesic derived from thebaine, used primarily for severe pain management when other treatments fail.\" \/>\n<meta name=\"robots\" content=\"index, follow, max-snippet:-1, max-image-preview:large, max-video-preview:-1\" \/>\n<link rel=\"canonical\" href=\"https:\/\/bristishpharmacy.co.uk\/es\/shop\/oximorfona\/\" \/>\n<meta property=\"og:locale\" content=\"es_ES\" \/>\n<meta property=\"og:type\" content=\"article\" \/>\n<meta property=\"og:title\" content=\"Oxymorphon\" \/>\n<meta property=\"og:description\" content=\"Oxymorphone is a potent, semi-synthetic pure opioid agonist derived from morphine. 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