{"id":7094,"date":"2025-12-14T15:43:50","date_gmt":"2025-12-14T15:43:50","guid":{"rendered":"https:\/\/bristishpharmacy.co.uk\/?post_type=product&p=7094"},"modified":"2026-09-16T09:54:42","modified_gmt":"2026-09-16T09:54:42","slug":"fentanyl-aurobindo","status":"publish","type":"product","link":"https:\/\/bristishpharmacy.co.uk\/nl\/shop\/fentanyl-aurobindo\/","title":{"rendered":"Fentanyl Aurobindo"},"content":{"rendered":"

\u00a0<\/h1>\n
\n
\n

1. Classification and Chemical Overview<\/h2>\n

Fentanyl is a potent synthetic phenylpiperidine-derivative opioid analgesic. Under the Anatomical Therapeutic Chemical (ATC) classification system, it is indexed under N02AB03.<\/p>\n

Chemically designated as N<\/span><\/span><\/span><\/span><\/span>-(1-phenethylpiperidin-4-yl)-N<\/span><\/span><\/span><\/span><\/span>-phenylpropanamide, fentanyl is a highly lipophilic compound structured for efficient transdermal delivery across the stratum corneum. In United Kingdom clinical practice, Fentanyl Aurobindo is presented as transdermal patches delivering active substance at precise nominal release rates over a 72-hour period (available in strengths including 12, 25, 50, 75, and 100 micrograms\/hour). Under the Human Medicines Regulations 2012, fentanyl is classified as a Prescription Only Medicine (POM) and is controlled under Schedule 2 of the Misuse of Drugs Regulations 2001 (as amended).<\/p>\n

2. Mechanism of Action and Pharmacodynamics<\/h2>\n

Fentanyl acts as a selective, high-affinity agonist at central and peripheral \u03bc<\/span><\/span><\/span><\/span><\/span>-opioid receptors. It displays minimal binding affinity for \u03ba<\/span><\/span><\/span><\/span><\/span>– and \u03b4<\/span><\/span><\/span><\/span><\/span>-opioid receptor subtypes.<\/p>\n

Binding to the membrane-bound G-protein-coupled \u03bc<\/span><\/span><\/span><\/span><\/span>-opioid receptor inhibits adenylyl cyclase activity, reducing intracellular cyclic AMP (cAMP) levels. This inhibition prompts the closure of voltage-gated presynaptic calcium channels and the opening of inwardly rectifying postsynaptic potassium channels. Consequently, primary afferent nociceptors undergo hyperpolarisation, preventing the presynaptic release of excitatory neurotransmitters\u2014including substance P and glutamate\u2014within the dorsal horn of the spinal cord and ascending thalamic pathways.<\/p>\n

Systemic physiological actions encompass central analgesia, sedation, mood alteration, peripheral vasodilation, dose-dependent depression of the medullary respiratory center, and suppression of the cough reflex. The analgesic efficacy scales directly with plasma concentration, as does the risk of severe opioid-induced toxicity.<\/p>\n

3. Approved UK Clinical Indications and Therapeutic Scope<\/h2>\n

Licensing by the Medicines and Healthcare products Regulatory Agency (MHRA) for transdermal fentanyl includes:<\/p>\n

    \n
  • \n

    Severe Chronic Pain (Adults):<\/b> Long-term management of severe chronic pain that can be adequately managed only with strong opioid analgesics in opioid-tolerant adult patients.<\/p>\n<\/li>\n

  • \n

    Severe Chronic Intractable Pain (Paediatrics):<\/b> Long-term management of severe chronic pain in opioid-tolerant paediatric patients aged 2 years and older who are already receiving opioid therapy.<\/p>\n<\/li>\n<\/ul>\n

    Opioid Tolerance Requirement:<\/i> Transdermal fentanyl patches are strictly contraindicated in opioid-naive individuals due to the high risk of fatal hypoventilation. Opioid tolerance is defined in clinical guidelines as receiving at least 60 mg oral morphine daily (or an equianalgesic dosage of an alternative opioid) continuously for one week or longer.<\/p>\n

    NICE & BNF Guidance Context:<\/i> National Institute for Health and Care Excellence (NICE) guidelines place transdermal fentanyl as an alternative strong opioid option for stable, severe chronic pain (such as malignant cancer pain) in patients who experience intractable, dose-limiting side effects (e.g., severe constipation or nausea) from oral morphine, or who present with severe dysphagia or gastrointestinal malabsorption. It is not licensed or indicated for acute, subacute, or post-operative pain management.<\/p>\n

    4. Pharmacokinetic Profile and Metabolic Fate<\/h2>\n
      \n
    • \n

      Absorption:<\/b> Following application to intact skin, fentanyl is absorbed continuously through the epidermis. A cutaneous depot is established within the upper dermal layers before systemic vascular absorption proceeds. Minimal therapeutic concentrations are detectable within 6 to 12 hours of initial application, reaching steady-state peak plasma concentrations (T<\/span>ma<\/span>x<\/span><\/span><\/span><\/span><\/span>\u200b<\/span><\/span><\/span><\/span><\/span><\/span><\/span><\/span><\/span>) between 24 and 72 hours.<\/p>\n<\/li>\n

    • \n

      Distribution:<\/b> Fentanyl distributes rapidly into parenchymal tissues, exhibiting a large apparent volume of distribution (V<\/span>d<\/span><\/span><\/span><\/span>\u200b<\/span><\/span><\/span><\/span><\/span><\/span><\/span><\/span><\/span>) ranging from 3<\/span>\u00a0to\u00a0<\/span><\/span>10<\/span>\u00a0L\/kg<\/span><\/span><\/span><\/span><\/span><\/span>. Plasma protein binding ranges from 80% to 85%, bound predominantly to \u03b1<\/span>1<\/span><\/span><\/span><\/span>\u200b<\/span><\/span><\/span><\/span><\/span><\/span><\/span><\/span><\/span>-acid glycoprotein and albumin. Fentanyl readily crosses the blood-brain barrier, placental barrier, and partition into human breast milk.<\/p>\n<\/li>\n

    • \n

      Biotransformation:<\/b> Fentanyl undergoes extensive hepatic clearance via Phase I N<\/span><\/span><\/span><\/span><\/span>-dealkylation, mediated almost exclusively by Cytochrome P450 3A4 (CYP3A4) isoenzymes, yielding the primary metabolite norfentanyl. Norfentanyl, alongside other minor oxidative degradation products, is pharmacologically inactive.<\/p>\n<\/li>\n

    • \n

      Elimination:<\/b> Within 72 to 96 hours post-application, approximately 75% of the dose is excreted in urine (predominantly as inactive metabolites, with less than 10% excreted as unchanged drug) and 9% in faeces. Following patch removal, continuous release from the cutaneous depot maintains serum levels; plasma concentrations decline gradually, displaying a mean terminal elimination half-life (t<\/span>1\/2<\/span><\/span><\/span><\/span>\u200b<\/span><\/span><\/span><\/span><\/span><\/span><\/span><\/span><\/span>) of 17 to 25 hours.<\/p>\n<\/li>\n<\/ul>\n

      5. Physiological Effects and Adverse Event Spectrum<\/h2>\n

      Transdermal fentanyl modifies ascending pain pathways while altering autonomic, respiratory, and central nervous system dynamics.<\/p>\n

      Adverse Drug Reaction Spectrum<\/h3>\n
        \n
      • \n

        Very Common (\u2265<\/span><\/span>1\/10<\/span><\/span><\/span><\/span><\/span>):<\/b> Somnolence, dizziness, headache, nausea, vomiting, constipation, hyperhidrosis, pruritus.<\/p>\n<\/li>\n

      • \n

        Common (\u2265<\/span><\/span>1\/100<\/span><\/span><\/span><\/span><\/span> to <<\/span><\/span>1\/10<\/span><\/span><\/span><\/span><\/span>):<\/b> Hypersensitivity reactions, anorexia, insomnia, depression, anxiety, confusion, hallucinations, tremor, paraesthesia, vertigo, palpitations, tachycardia, hypertension, dyspnoea, diarrhoea, dry mouth, abdominal pain, dyspepsia, rash, application site erythema, muscle spasms, urinary retention, fatigue, peripheral oedema, asthenia.<\/p>\n<\/li>\n

      • \n

        Uncommon (\u2265<\/span><\/span>1\/1<\/span>,<\/span>000<\/span><\/span><\/span><\/span><\/span> to <<\/span><\/span>1\/100<\/span><\/span><\/span><\/span><\/span>):<\/b> Agitation, disorientation, euphoria, hypoaesthesia, convulsions, bradycardia, cyanosis, hypotension, respiratory depression, paralytic ileus, eczema, dermatitis, application site reactions, erectile dysfunction, body temperature fluctuations.<\/p>\n<\/li>\n

      • \n

        Rare (\u2265<\/span><\/span>1\/10<\/span>,<\/span>000<\/span><\/span><\/span><\/span><\/span> to <<\/span><\/span>1\/1<\/span>,<\/span>000<\/span><\/span><\/span><\/span><\/span>):<\/b> Miosis, apnoea, hypoventilation, subileus, application site vesicles.<\/p>\n<\/li>\n

      • \n

        Frequency Unknown:<\/b> Anaphylactic shock, androgen deficiency, secondary adrenal insufficiency, physical dependence, drug withdrawal syndrome, neonatal withdrawal syndrome.<\/p>\n<\/li>\n<\/ul>\n

        6. Contraindications, Drug Interactions, and Clinical Precautions<\/h2>\n

        Contra-indicaties<\/h3>\n
          \n
        • \n

          Hypersensitivity to fentanyl or patch adhesive excipients.<\/p>\n<\/li>\n

        • \n

          Opioid-naive status (high risk of life-threatening respiratory depression).<\/p>\n<\/li>\n

        • \n

          Acute pain, post-operative pain, or short-term pain conditions requiring rapid titration.<\/p>\n<\/li>\n

        • \n

          Severe respiratory depression, acute lung disease, or severe obstructive airways disease.<\/p>\n<\/li>\n<\/ul>\n

          Key Drug Interactions<\/h3>\n
            \n
          • \n

            CYP3A4 Inhibitors:<\/b> Co-administration with strong or moderate CYP3A4 inhibitors (e.g., ketoconazole, itraconazole, ritonavir, clarithromycin, erythromycin, diltiazem, grapefruit juice) reduces fentanyl clearance, elevating systemic exposure and escalating the risk of fatal respiratory depression.<\/p>\n<\/li>\n

          • \n

            CYP3A4 Inducers:<\/b> Concomitant use with CYP3A4 inducers (e.g., rifampicin, carbamazepine, phenytoin, St John’s Wort) accelerates clearance, risking reduced analgesic efficacy and precipitating withdrawal symptoms in opioid-dependent patients.<\/p>\n<\/li>\n

          • \n

            CNS Depressants & Alcohol:<\/b> Concurrent use with benzodiazepines, hypnotics, sedatives, general anaesthetics, phenothiazines, other opioids, or alcohol produces additive CNS and respiratory depression, hypotension, and potential coma.<\/p>\n<\/li>\n

          • \n

            Serotonergic Agents & MAOIs:<\/b> Concomitant administration with serotonergic agents (SSRIs, SNRIs) increases the risk of Serotonin Syndrome. Use with monoamine oxidase inhibitors (MAOIs) or within 14 days of their discontinuation is strictly contraindicated.<\/p>\n<\/li>\n<\/ul>\n

            Clinical Precautions and Monitoring<\/h3>\n
              \n
            • \n

              Accidental Exposure & Disposal:<\/b> Accidental transfer to non-patients or accidental ingestion by children carries fatal consequences. Patches must be folded immediately upon removal (adhesive sides pressed together) and disposed of securely.<\/p>\n<\/li>\n

            • \n

              External Heat & Fever:<\/b> Increased skin temperature enhances transdermal permeability and local cutaneous blood flow, dramatically accelerating fentanyl absorption. Patients must avoid exposing the application site to direct heat sources (e.g., heating pads, saunas, hot tubs, electric blankets). Patients developing high fever must be closely monitored for signs of opioid toxicity.<\/p>\n<\/li>\n

            • \n

              Respiratory Monitoring & Dose Tapering:<\/b> Baseline respiratory status, level of consciousness, and bowel function require systematic assessment. Abrupt discontinuation is contraindicated due to severe withdrawal phenomena; structured dose-tapering regimes must be implemented.<\/p>\n<\/li>\n

            • \n

              Organ Impairment:<\/b> Patients with hepatic or renal impairment exhibit reduced systemic clearance and require lower dosages alongside enhanced monitoring for toxicity.<\/p>\n<\/li>\n<\/ul>\n<\/div>\n<\/div>\n\n

              \n\n\t\t\n
                \n\t\t\t <\/ul>\n <\/div>","protected":false},"excerpt":{"rendered":"

                Fentanyl Aurobindo transdermal patches are potent opioid analgesics indicated for the management of severe chronic pain requiring continuous opioid therapy.<\/p>\n

                \u00a0<\/h3>","protected":false},"featured_media":7100,"comment_status":"closed","ping_status":"closed","template":"","meta":{"postBodyCss":"","postBodyMargin":[],"postBodyPadding":[],"postBodyBackground":{"backgroundType":"classic","gradient":""}},"product_brand":[],"product_cat":[107],"product_tag":[148],"class_list":["post-7094","product","type-product","status-publish","has-post-thumbnail","product_cat-painkillers","product_tag-fentanyl-aurobindo","instock","shipping-taxable","purchasable","product-type-simple"],"yoast_head":"\nFentanyl Aurobindo - British Pharmacy<\/title>\n<meta name=\"description\" content=\"Fentanyl Aurobindo is a potent opioid medication designed for the management of severe chronic pain in patients who require continuous.\" \/>\n<meta name=\"robots\" content=\"index, follow, max-snippet:-1, max-image-preview:large, max-video-preview:-1\" \/>\n<link rel=\"canonical\" href=\"https:\/\/bristishpharmacy.co.uk\/nl\/shop\/fentanyl-aurobindo\/\" \/>\n<meta property=\"og:locale\" content=\"nl_NL\" \/>\n<meta property=\"og:type\" content=\"article\" \/>\n<meta property=\"og:title\" content=\"Fentanyl Aurobindo\" \/>\n<meta property=\"og:description\" content=\"Fentanyl Aurobindo transdermal patches are potent opioid analgesics indicated for the management of severe chronic pain requiring continuous opioid therapy.\u00a0\" \/>\n<meta property=\"og:url\" content=\"https:\/\/bristishpharmacy.co.uk\/nl\/shop\/fentanyl-aurobindo\/\" \/>\n<meta property=\"og:site_name\" content=\"British Pharmacy\" \/>\n<meta property=\"article:modified_time\" content=\"2026-09-16T09:54:42+00:00\" \/>\n<meta property=\"og:image\" content=\"https:\/\/bristishpharmacy.co.uk\/wp-content\/uploads\/2025\/12\/5041968916481116958.jpg\" \/>\n\t<meta property=\"og:image:width\" content=\"1200\" \/>\n\t<meta property=\"og:image:height\" content=\"1200\" \/>\n\t<meta property=\"og:image:type\" content=\"image\/jpeg\" \/>\n<meta name=\"twitter:card\" content=\"summary_large_image\" \/>\n<meta name=\"twitter:label1\" content=\"Geschatte leestijd\" \/>\n\t<meta name=\"twitter:data1\" content=\"3 minuten\" \/>\n<script type=\"application\/ld+json\" class=\"yoast-schema-graph\">{\"@context\":\"https:\\\/\\\/schema.org\",\"@graph\":[{\"@type\":\"WebPage\",\"@id\":\"https:\\\/\\\/bristishpharmacy.co.uk\\\/shop\\\/fentanyl-aurobindo\\\/\",\"url\":\"https:\\\/\\\/bristishpharmacy.co.uk\\\/shop\\\/fentanyl-aurobindo\\\/\",\"name\":\"Fentanyl Aurobindo - 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